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Tytuł :
Use of telemedicine during the COVID-19 pandemic in patients with inflammatory arthritis: a retrospective study on feasibility and impact on patient-reported outcomes in a real-life setting.
Autorzy :
Chevallard M; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy. .
Belloli L; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Ughi N; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Adinolfi A; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Casu C; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Di Cicco M; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Filippini DA; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Muscarà M; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Schito E; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Verduci E; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Gentile MG; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Di Rosa B; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Zoppini L; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Carli C; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Destefani C; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Luisi A; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Vincenti EM; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
Epis OM; Division of Rheumatology, Multispecialist Medical Department, ASST Grande Ospedale Metropolitano Niguarda, Milan, Italy.
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Źródło :
Rheumatology international [Rheumatol Int] 2021 Jul; Vol. 41 (7), pp. 1253-1261. Date of Electronic Publication: 2021 May 04.
Typ publikacji :
Journal Article
MeSH Terms :
Patient Reported Outcome Measures*
SARS-CoV-2*
Telemedicine*
Arthritis/*drug therapy
COVID-19/*epidemiology
Adult ; Aged ; Arthritis, Psoriatic/drug therapy ; Arthritis, Rheumatoid/drug therapy ; Feasibility Studies ; Female ; Humans ; Male ; Middle Aged ; Outcome Assessment, Health Care ; Retrospective Studies ; Spondylitis, Ankylosing/drug therapy
Czasopismo naukowe
Tytuł :
The impact of different (rheumatoid) arthritis phenotypes on patients' lives.
Autorzy :
Luurssen-Masurel N; Department of Rheumatology, Erasmus Medical Center, Rotterdam, The Netherlands.
Weel AEAM; Department of Rheumatology, Erasmus Medical Center, Rotterdam, The Netherlands.; Department of Rheumatology, Maasstad Hospital, Rotterdam, The Netherlands.; Erasmus School of Health Policy and Management, Rotterdam, The Netherlands.
Hazes JMW; Department of Rheumatology, Erasmus Medical Center, Rotterdam, The Netherlands.
de Jong PHP; Department of Rheumatology, Erasmus Medical Center, Rotterdam, The Netherlands.
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Corporate Authors :
tREACH group investigators
Źródło :
Rheumatology (Oxford, England) [Rheumatology (Oxford)] 2021 Aug 02; Vol. 60 (8), pp. 3716-3726.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Activities of Daily Living*
Arthritis, Rheumatoid/*physiopathology
Adult ; Aged ; Arthritis/immunology ; Arthritis/physiopathology ; Arthritis, Rheumatoid/immunology ; Autoantibodies/immunology ; Cost of Illness ; Female ; Humans ; Male ; Middle Aged ; Minimal Clinically Important Difference ; Patient Reported Outcome Measures ; Phenotype ; Quality of Life
Czasopismo naukowe
Tytuł :
Comparative evaluation of the protective effects of oral administration of auraptene and umbelliprenin against CFA-induced chronic inflammation with polyarthritis in rats.
Autorzy :
Shirani K; Department of Toxicology, Faculty of Medical Sciences, Tarbiat Modares University, Tehran, Iran.
Iranshahi M; Biotechnology Research Center, Pharmaceutical Technology Institute, Mashhad University of Medical Sciences, Mashhad, Iran.
Askari VR; Pharmacological Research Center of Medicinal Plants, Mashhad University of Medical Sciences, Mashhad, Iran; Department of Persian Medicine, School of Persian and Complementary Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Gholizadeh Z; School of Pharmacy, Mashhad University of Medical Sciences, Mashhad, Iran.
Zadeh AA; Department of Medical Genetics, Faculty of Medicines, Mashhad University of Medical Sciences, Mashhad, Iran.
Zeinali M; School of Pharmacy, Mashhad University of Medical Sciences, Mashhad, Iran.
Hassani FV; Mashhad University of Medical Sciences, Mashhad, Iran.
Taherzadeh Z; Targeted Drug Delivery Research Center, Mashhad University of Medical Sciences, P.O. Box: 1365-91775, Mashhad, Iran. Electronic address: .
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Źródło :
Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie [Biomed Pharmacother] 2021 Jul; Vol. 139, pp. 111635. Date of Electronic Publication: 2021 May 07.
Typ publikacji :
Comparative Study; Journal Article
MeSH Terms :
Arthritis/*drug therapy
Coumarins/*pharmacology
Freund's Adjuvant/*pharmacology
Inflammation/*chemically induced
Inflammation/*drug therapy
Protective Agents/*pharmacology
Umbelliferones/*pharmacology
Administration, Oral ; Animals ; Anti-Inflammatory Agents/pharmacology ; Arthritis/metabolism ; Arthritis, Experimental/chemically induced ; Arthritis, Experimental/drug therapy ; Arthritis, Experimental/metabolism ; Arthritis, Rheumatoid/chemically induced ; Arthritis, Rheumatoid/drug therapy ; Arthritis, Rheumatoid/metabolism ; Cytokines/metabolism ; Edema/chemically induced ; Edema/drug therapy ; Edema/metabolism ; Inflammation/metabolism ; Male ; Rats ; Rats, Wistar
Czasopismo naukowe
Tytuł :
Inhibition of lipid phosphatase SHIP1 expands myeloid-derived suppressor cells and attenuates rheumatoid arthritis in mice.
Autorzy :
So EY; Division of Hematology/Oncology, Department of Medicine, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, Rhode Island.
Sun C; Division of Rheumatology, Department of Medicine, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, Rhode Island.
Wu KQ; Division of Hematology/Oncology, Department of Medicine, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, Rhode Island.
Dubielecka PM; Division of Hematology/Oncology, Department of Medicine, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, Rhode Island.
Reginato AM; Division of Rheumatology, Department of Medicine, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, Rhode Island.
Liang OD; Division of Hematology/Oncology, Department of Medicine, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence, Rhode Island.
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Źródło :
American journal of physiology. Cell physiology [Am J Physiol Cell Physiol] 2021 Sep 01; Vol. 321 (3), pp. C569-C584. Date of Electronic Publication: 2021 Jul 21.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
MeSH Terms :
Anti-Inflammatory Agents/*pharmacology
Arthritis, Experimental/*drug therapy
Cholestanes/*pharmacology
Myeloid-Derived Suppressor Cells/*immunology
Phosphatidylinositol-3,4,5-Trisphosphate 5-Phosphatases/*genetics
T-Lymphocytes, Regulatory/*immunology
Adoptive Transfer ; Animals ; Arthritis, Experimental/genetics ; Arthritis, Experimental/immunology ; Arthritis, Experimental/pathology ; Arthritis, Rheumatoid/genetics ; Arthritis, Rheumatoid/immunology ; Arthritis, Rheumatoid/pathology ; Cell Communication ; Cell Differentiation/drug effects ; Cell Proliferation/drug effects ; Gene Expression ; Humans ; Joint Capsule/drug effects ; Joint Capsule/immunology ; Joint Capsule/pathology ; Mice ; Mice, Inbred DBA ; Mice, Knockout ; Myeloid-Derived Suppressor Cells/cytology ; Myeloid-Derived Suppressor Cells/transplantation ; Phosphatidylinositol-3,4,5-Trisphosphate 5-Phosphatases/antagonists & inhibitors ; Phosphatidylinositol-3,4,5-Trisphosphate 5-Phosphatases/immunology ; Severity of Illness Index ; T-Lymphocytes, Regulatory/drug effects ; T-Lymphocytes, Regulatory/pathology
Czasopismo naukowe
Tytuł :
Reactive arthritis after COVID-19.
Autorzy :
Hønge BL; Department of Infectious Diseases, Aarhus Universitet, Aarhus, Denmark .
Hermansen MF; Department of Internal Medicine, Randers Regional Hospital, Randers, Midtjylland, Denmark.
Storgaard M; Intensive Care, Aarhus Universitetshospital, Aarhus, Denmark.
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Źródło :
BMJ case reports [BMJ Case Rep] 2021 Mar 02; Vol. 14 (3). Date of Electronic Publication: 2021 Mar 02.
Typ publikacji :
Case Reports; Journal Article
MeSH Terms :
Arthritis*/drug therapy
Arthritis*/etiology
Arthritis*/physiopathology
Arthritis, Reactive*/diagnosis
Arthritis, Reactive*/drug therapy
Arthritis, Reactive*/physiopathology
Arthritis, Reactive*/virology
COVID-19*/complications
COVID-19*/physiopathology
COVID-19*/therapy
Synovial Fluid*/cytology
Synovial Fluid*/immunology
Anti-Inflammatory Agents, Non-Steroidal/*administration & dosage
Prednisolone/*administration & dosage
Anti-Inflammatory Agents/administration & dosage ; Arthralgia/diagnosis ; Arthralgia/etiology ; Arthritis, Rheumatoid/diagnosis ; Autoantibodies/analysis ; Diagnosis, Differential ; Humans ; Knee Joint/diagnostic imaging ; Lower Extremity/pathology ; Male ; Middle Aged ; Radiography/methods ; Treatment Outcome
Czasopismo naukowe
Tytuł :
Mediation of Interleukin-23 and Tumor Necrosis Factor-Driven Reactive Arthritis by Chlamydia-Infected Macrophages in SKG Mice.
Autorzy :
Romand X; Université Grenoble Alpes, GREPI TIMC-IMAG, UMR 5525, Grenoble, France.
Liu X; University of Queensland Diamantina Institute and Princess Alexandra Hospital, Brisbane, Queensland, Australia.
Rahman MA; University of Queensland Diamantina Institute and Princess Alexandra Hospital, Brisbane, Queensland, Australia.
Bhuyan ZA; University of Queensland Diamantina Institute and Princess Alexandra Hospital, Brisbane, Queensland, Australia, and North South University, Dhaka, Bangladesh.
Douillard C; Université Grenoble Alpes, GREPI TIMC-IMAG, UMR 5525, Grenoble, France.
Kedia RA; University of Queensland Diamantina Institute and Princess Alexandra Hospital, Brisbane, Queensland, Australia.
Stone N; University of Queensland Diamantina Institute and Princess Alexandra Hospital, Brisbane, Queensland, Australia.
Roest D; University of Queensland Diamantina Institute and Princess Alexandra Hospital, Brisbane, Queensland, Australia.
Chew ZH; University of Queensland Diamantina Institute and Princess Alexandra Hospital, Brisbane, Queensland, Australia.
Cameron AJ; University of Queensland Diamantina Institute and Princess Alexandra Hospital, Brisbane, Queensland, Australia.
Rehaume LM; University of Queensland Diamantina Institute and Princess Alexandra Hospital, Brisbane, Queensland, Australia.
Bozon A; Université Grenoble Alpes, GREPI TIMC-IMAG, UMR 5525, Grenoble, France.
Habib M; Université Grenoble Alpes, GREPI TIMC-IMAG, UMR 5525, Grenoble, France.
Armitage CW; Queensland University of Technology, Brisbane, Queensland, Australia, and King's College London, London, UK.
Nguyen MVC; Université Grenoble Alpes, GREPI TIMC-IMAG, UMR 5525, Grenoble, France.
Favier B; Université Grenoble Alpes, GREPI TIMC-IMAG, UMR 5525, Grenoble, France.
Beagley K; Queensland University of Technology, Brisbane, Queensland, Australia.
Maurin M; Université Grenoble Alpes, GREPI TIMC-IMAG, UMR 5525, Grenoble, France.
Gaudin P; Université Grenoble Alpes, GREPI TIMC-IMAG, UMR 5525, Grenoble, France.
Thomas R; University of Queensland Diamantina Institute and Princess Alexandra Hospital, Brisbane, Queensland, Australia.
Wells TJ; University of Queensland Diamantina Institute and Princess Alexandra Hospital, Brisbane, Queensland, Australia.
Baillet A; Université Grenoble Alpes, GREPI TIMC-IMAG, UMR 5525, Grenoble, France.
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Źródło :
Arthritis & rheumatology (Hoboken, N.J.) [Arthritis Rheumatol] 2021 Jul; Vol. 73 (7), pp. 1200-1210. Date of Electronic Publication: 2021 May 28.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Arthritis, Reactive/*immunology
Chlamydia Infections/*immunology
Interleukin-23/*immunology
Interleukin-23 Subunit p19/*immunology
Macrophages/*immunology
Tumor Necrosis Factor-alpha/*immunology
Animals ; Arthritis, Experimental/genetics ; Arthritis, Reactive/genetics ; Chlamydia muridarum ; Female ; Gene Expression Profiling ; Heat-Shock Proteins/genetics ; Heat-Shock Proteins/immunology ; Interleukin-12 Subunit p40/genetics ; Interleukin-12 Subunit p40/immunology ; Interleukin-17/genetics ; Interleukin-17/immunology ; Interleukin-23 Subunit p19/genetics ; Macrophages/microbiology ; Mice ; Mice, Inbred BALB C ; Monomeric GTP-Binding Proteins/genetics ; Monomeric GTP-Binding Proteins/immunology ; Tumor Necrosis Factor-alpha/genetics ; ZAP-70 Protein-Tyrosine Kinase/genetics
Czasopismo naukowe
Tytuł :
Alantolactone alleviates collagen-induced arthritis and inhibits Th17 cell differentiation through modulation of STAT3 signalling.
Autorzy :
Chen HL; Department of Surgery, Division of Urology, Tung's Taichung MetroHarbor Hospital, Taichung, Taiwan, ROC.; Program in Translational Medicine, National Chung Hsing University, Taichung, Taiwan, ROC.
Lin SC; Bachelor Degree Program in Marine Biotechnology, College of Life Sciences, National Taiwan Ocean University, Keelung, Taiwan, ROC.
Li S; College of Chemistry & Chemical Engineering, Hubei Key Laboratory for Processing & Application of Catalytic Materials, Huanggang Normal University, Huanggang, PR China.
Tang KT; Program in Translational Medicine, National Chung Hsing University, Taichung, Taiwan, ROC.; Faculty of Medicine, National Yang-Ming University, Taipei, Taiwan, ROC.; Division of Allergy, Immunology and Rheumatology, Taichung Veterans General Hospital, Taichung, Taiwan, ROC.
Lin CC; Program in Translational Medicine, National Chung Hsing University, Taichung, Taiwan, ROC.; Institute of Biomedical Science, The iEGG and Animal Biotechnology Center, National Chung-Hsing University, Taichung, Taiwan, ROC.; Department of Medical Research, China Medical University Hospital, Taichung, Taiwan, ROC.; Department of Medical Research, Taichung Veterans General Hospital, Taichung, Taiwan, ROC.; Department of Pharmacology, College of Medicine, Kaohsiung Medical University, Kaohsiung, Taiwan, ROC.
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Źródło :
Pharmaceutical biology [Pharm Biol] 2021 Dec; Vol. 59 (1), pp. 134-145.
Typ publikacji :
Journal Article
MeSH Terms :
Anti-Inflammatory Agents/*pharmacology
Arthritis, Experimental/*drug therapy
Lactones/*pharmacology
Sesquiterpenes, Eudesmane/*pharmacology
Th17 Cells/*drug effects
Animals ; Anti-Inflammatory Agents/administration & dosage ; Anti-Inflammatory Agents/isolation & purification ; Arthritis, Experimental/pathology ; Arthritis, Rheumatoid/drug therapy ; Arthritis, Rheumatoid/pathology ; Cell Differentiation/drug effects ; Cytokines ; Dose-Response Relationship, Drug ; Female ; Immunologic Factors/administration & dosage ; Immunologic Factors/isolation & purification ; Immunologic Factors/pharmacology ; Inula/chemistry ; Lactones/administration & dosage ; Lactones/isolation & purification ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Inbred DBA ; STAT3 Transcription Factor/metabolism ; Sesquiterpenes, Eudesmane/administration & dosage ; Sesquiterpenes, Eudesmane/isolation & purification ; Signal Transduction/drug effects ; Th17 Cells/cytology
Czasopismo naukowe
Tytuł :
The role of interleukin-6 trans-signalling on cardiovascular dysfunction in inflammatory arthritis.
Autorzy :
Davies R; CREATE Centre, Division of Infection and Immunity, , Cardiff, UK.
Williams J; CREATE Centre, Division of Infection and Immunity, , Cardiff, UK.
Sime K; CREATE Centre, Division of Infection and Immunity, , Cardiff, UK.
Jin HS; CREATE Centre, Division of Infection and Immunity, , Cardiff, UK.
Thompson C; CREATE Centre, Division of Infection and Immunity, , Cardiff, UK.
Jordan L; CREATE Centre, Division of Infection and Immunity, , Cardiff, UK.
Lang D; Division of Medical Education, Cardiff University School of Medicine, Cardiff, UK.
Halcox JP; Institute of Life Sciences, Swansea University, Swansea, UK.
Ellins E; Institute of Life Sciences, Swansea University, Swansea, UK.
Jones GW; School of Cellular and Molecular Medicine, Biomedical Sciences Building, University of Bristol, Bristol, UK.
Jones SA; CREATE Centre, Division of Infection and Immunity, , Cardiff, UK.
Rose-John S; Institute of Biochemistry, Christian-Albrecht Universität, Kiel, Germany.
Williams A; CREATE Centre, Division of Infection and Immunity, , Cardiff, UK.
Choy E; CREATE Centre, Division of Infection and Immunity, , Cardiff, UK.
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Źródło :
Rheumatology (Oxford, England) [Rheumatology (Oxford)] 2021 Jun 18; Vol. 60 (6), pp. 2852-2861.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Arthritis, Rheumatoid/*drug therapy
Cardiovascular Diseases/*drug therapy
Etanercept/*pharmacology
Interleukin-6/*metabolism
Recombinant Fusion Proteins/*pharmacology
Vascular Cell Adhesion Molecule-1/*metabolism
Animals ; Antirheumatic Agents/pharmacology ; Arthritis, Experimental ; Arthritis, Rheumatoid/complications ; Biomarkers/metabolism ; Cardiovascular Diseases/etiology ; Cross-Sectional Studies ; Disease Models, Animal ; Female ; Humans ; Male ; Mice ; Middle Aged
Czasopismo naukowe
Tytuł :
Phytochemical and In-Vivo Anti-Arthritic Significance of Aloe thraskii Baker in Combined Therapy with Methotrexate in Adjuvant-Induced Arthritis in Rats.
Autorzy :
Kamal RM; Department of Pharmacognosy, Faculty of Pharmacy, Cairo University, Cairo 11562, Egypt.
Sabry MM; Department of Pharmacognosy, Faculty of Pharmacy, Cairo University, Cairo 11562, Egypt.
Aly ZY; Department of Biochemistry, National Organization for Drug Control and Research (NODCAR), Giza 35521, Egypt.
Hifnawy MS; Department of Pharmacognosy, Faculty of Pharmacy, Cairo University, Cairo 11562, Egypt.
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Źródło :
Molecules (Basel, Switzerland) [Molecules] 2021 Jun 15; Vol. 26 (12). Date of Electronic Publication: 2021 Jun 15.
Typ publikacji :
Journal Article
MeSH Terms :
Aloe/*metabolism
Arthritis/*drug therapy
Plant Extracts/*pharmacology
Animals ; Antioxidants/pharmacology ; Antirheumatic Agents/therapeutic use ; Arthritis, Experimental/drug therapy ; Arthritis, Rheumatoid/drug therapy ; Freund's Adjuvant/adverse effects ; Male ; Methotrexate/pharmacology ; Oxidative Stress/drug effects ; Phytochemicals/pharmacology ; Plant Leaves/metabolism ; Rats ; Rats, Wistar
Czasopismo naukowe
Tytuł :
Inhibition of regulator of G protein signaling 10, aggravates rheumatoid arthritis progression by promoting NF-κB signaling pathway.
Autorzy :
Ren J; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, People's Republic of China.
Wei W; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, People's Republic of China.
Tan L; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, People's Republic of China.
Yang Q; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, People's Republic of China.
Lu Q; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, People's Republic of China.
Ding H; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, People's Republic of China.
Yue Y; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, People's Republic of China.
Tian Y; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, People's Republic of China.
Hao L; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, People's Republic of China.
Wang M; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, People's Republic of China. Electronic address: .
Li J; The State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases, Department of Prosthodontics, West China Hospital of Stomatology, Sichuan University, Sichuan, People's Republic of China. Electronic address: .
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Źródło :
Molecular immunology [Mol Immunol] 2021 Jun; Vol. 134, pp. 236-246. Date of Electronic Publication: 2021 Apr 06.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Arthritis, Rheumatoid/*immunology
NF-kappa B/*metabolism
RGS Proteins/*immunology
Signal Transduction/*immunology
Animals ; Arthritis, Experimental/immunology ; Arthritis, Experimental/metabolism ; Arthritis, Rheumatoid/metabolism ; Disease Progression ; Mice ; Mice, Inbred DBA ; RAW 264.7 Cells ; RGS Proteins/metabolism
Czasopismo naukowe
Tytuł :
Monocytic MDSCs skew Th17 cells toward a pro-osteoclastogenic phenotype and potentiate bone erosion in rheumatoid arthritis.
Autorzy :
Chen S; Department of Traditional Chinese Internal Medicine, School of Traditional Chinese Medicine.; Department of Rheumatic & TCM Medical Center, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Guo C; Department of Human & Molecular Genetics.; Institute of Molecular Medicine, Virginia Commonwealth University School of Medicine, Richmond, VA, USA.
Wang R; Department of Traditional Chinese Internal Medicine, School of Traditional Chinese Medicine.; Department of Rheumatic & TCM Medical Center, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Feng Z; Department of Traditional Chinese Internal Medicine, School of Traditional Chinese Medicine.
Liu Z; Department of Human & Molecular Genetics.; Institute of Molecular Medicine, Virginia Commonwealth University School of Medicine, Richmond, VA, USA.
Wu L; Department of Traditional Chinese Internal Medicine, School of Traditional Chinese Medicine.
Zhao D; Department of Traditional Chinese Internal Medicine, School of Traditional Chinese Medicine.
Zheng S; Department of Traditional Chinese Internal Medicine, School of Traditional Chinese Medicine.
Chen F; Department of Traditional Chinese Internal Medicine, School of Traditional Chinese Medicine.
Zhang D; Department of Traditional Chinese Internal Medicine, School of Traditional Chinese Medicine.
Xu J; Department of Traditional Chinese Internal Medicine, School of Traditional Chinese Medicine.
Zhu J; Department of Rheumatic & TCM Medical Center, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Chen X; Guangdong Provincial Key Laboratory of Tropical Disease Research, Department of Pathogen Biology, Southern Medical University School of Public Health, Guangzhou.
Li Z; Department of Rheumatology and Immunology, Peking University People's Hospital & Beijing Key Laboratory for Rheumatism Mechanism and Immune Diagnosis, Beijing, China.
Wise CM; Department of Internal Medicine, Virginia Commonwealth University Medical Center.
Li J; Department of Traditional Chinese Internal Medicine, School of Traditional Chinese Medicine.; Department of Rheumatic & TCM Medical Center, Nanfang Hospital, Southern Medical University, Guangzhou, China.
Wang XY; Department of Human & Molecular Genetics.; Institute of Molecular Medicine, Virginia Commonwealth University School of Medicine, Richmond, VA, USA.; McGuire VA Medical Center, Richmond, VA, USA.
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Źródło :
Rheumatology (Oxford, England) [Rheumatology (Oxford)] 2021 May 14; Vol. 60 (5), pp. 2409-2420.
Typ publikacji :
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
MeSH Terms :
Arthritis, Rheumatoid/*immunology
Bone Resorption/*immunology
Monocytes/*immunology
Myeloid-Derived Suppressor Cells/*immunology
Osteoclasts/*immunology
Th17 Cells/*immunology
Animals ; Arthritis, Experimental/immunology ; Arthritis, Experimental/pathology ; Arthritis, Rheumatoid/pathology ; Bone Resorption/pathology ; Cell Differentiation/immunology ; Humans ; Mice ; Phenotype
Czasopismo naukowe
Tytuł :
Loss of α2-6 sialylation promotes the transformation of synovial fibroblasts into a pro-inflammatory phenotype in arthritis.
Autorzy :
Wang Y; Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, UK.
Khan A; Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, UK.
Antonopoulos A; Department of Life Sciences, Imperial College London, London, UK.
Bouché L; Department of Life Sciences, Imperial College London, London, UK.
Buckley CD; Rheumatology Research Group, Institute for Inflammation and Ageing, College of Medical and Dental Sciences, University of Birmingham, Queen Elizabeth Hospital, Birmingham, UK.; The Kennedy Institute of Rheumatology, University of Oxford, Oxford, UK.; Research into Inflammatory Arthritis Centre Versus Arthritis (RACE), Glasgow, Birmingham, Newcastle, Oxford, UK.
Filer A; Rheumatology Research Group, Institute for Inflammation and Ageing, College of Medical and Dental Sciences, University of Birmingham, Queen Elizabeth Hospital, Birmingham, UK.; Research into Inflammatory Arthritis Centre Versus Arthritis (RACE), Glasgow, Birmingham, Newcastle, Oxford, UK.
Raza K; Rheumatology Research Group, Institute for Inflammation and Ageing, College of Medical and Dental Sciences, University of Birmingham, Queen Elizabeth Hospital, Birmingham, UK.; Sandwell and West Birmingham Hospitals NHS Trust, Birmingham, UK.
Li KP; Institute of Chinese Medicinal Sciences, Guangdong Pharmaceutical University, Guangzhou, China.
Tolusso B; Research into Inflammatory Arthritis Centre Versus Arthritis (RACE), Glasgow, Birmingham, Newcastle, Oxford, UK.; Division of Rheumatology, Fondazione Policlinico Universitario A. Gemelli IRCCS, Rome, Italy.
Gremese E; Research into Inflammatory Arthritis Centre Versus Arthritis (RACE), Glasgow, Birmingham, Newcastle, Oxford, UK.; Division of Rheumatology, Fondazione Policlinico Universitario A. Gemelli IRCCS, Rome, Italy.
Kurowska-Stolarska M; Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, UK.; Research into Inflammatory Arthritis Centre Versus Arthritis (RACE), Glasgow, Birmingham, Newcastle, Oxford, UK.
Alivernini S; Research into Inflammatory Arthritis Centre Versus Arthritis (RACE), Glasgow, Birmingham, Newcastle, Oxford, UK.; Division of Rheumatology, Fondazione Policlinico Universitario A. Gemelli IRCCS, Rome, Italy.; Division of Rheumatology, Università Cattolica del Sacro Cuore, Rome, Italy.
Dell A; Department of Life Sciences, Imperial College London, London, UK.
Haslam SM; Department of Life Sciences, Imperial College London, London, UK.
Pineda MA; Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, UK. .; Research into Inflammatory Arthritis Centre Versus Arthritis (RACE), Glasgow, Birmingham, Newcastle, Oxford, UK. .
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Źródło :
Nature communications [Nat Commun] 2021 Apr 20; Vol. 12 (1), pp. 2343. Date of Electronic Publication: 2021 Apr 20.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Arthritis, Rheumatoid/*metabolism
Arthritis, Rheumatoid/*pathology
Sialic Acids/*metabolism
Synovial Membrane/*metabolism
Synovial Membrane/*pathology
Animals ; Arthritis, Experimental/pathology ; Arthritis, Rheumatoid/immunology ; Cell Line ; Cytokines/metabolism ; Down-Regulation ; Fibroblasts/immunology ; Fibroblasts/metabolism ; Fibroblasts/pathology ; Glycosylation ; Humans ; Inflammation/immunology ; Inflammation/metabolism ; Inflammation/pathology ; Male ; Metabolic Networks and Pathways ; Mice ; Mice, Inbred DBA ; Phenotype ; RNA-Seq ; Sialyltransferases/genetics ; Sialyltransferases/metabolism ; Synovial Membrane/immunology ; Transcriptome
Czasopismo naukowe
Tytuł :
[Mechanism of Xiaowugui decoction for treating collagen-induced arthritis in mice].
Autorzy :
Shan W; School of Traditional Chinese Medicine, Southern Medical University, Guangzhou 510515, China.
Zhu X; School of Traditional Chinese Medicine, Southern Medical University, Guangzhou 510515, China.
Qi J; School of Traditional Chinese Medicine, Southern Medical University, Guangzhou 510515, China.
Hu F; School of Traditional Chinese Medicine, Southern Medical University, Guangzhou 510515, China.
Li C; School of Traditional Chinese Medicine, Southern Medical University, Guangzhou 510515, China.
Nie X; School of Traditional Chinese Medicine, Southern Medical University, Guangzhou 510515, China.
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Źródło :
Nan fang yi ke da xue xue bao = Journal of Southern Medical University [Nan Fang Yi Ke Da Xue Xue Bao] 2020 Nov 30; Vol. 40 (11), pp. 1682-1688.
Typ publikacji :
Journal Article
MeSH Terms :
Arthritis, Experimental*/drug therapy
Animals ; Arthritis, Rheumatoid/drug therapy ; Male ; Methotrexate ; Mice ; Mice, Inbred DBA ; Th17 Cells
Czasopismo naukowe
Tytuł :
Lasp1 regulates adherens junction dynamics and fibroblast transformation in destructive arthritis.
Autorzy :
Beckmann D; Institute of Musculoskeletal Medicine, University Hospital Münster, Albert-Schweitzer-Campus 1, Building D3, Münster, Germany.
Römer-Hillmann A; Institute of Musculoskeletal Medicine, University Hospital Münster, Albert-Schweitzer-Campus 1, Building D3, Münster, Germany.
Krause A; Institute of Musculoskeletal Medicine, University Hospital Münster, Albert-Schweitzer-Campus 1, Building D3, Münster, Germany.
Hansen U; Institute of Musculoskeletal Medicine, University Hospital Münster, Albert-Schweitzer-Campus 1, Building D3, Münster, Germany.
Wehmeyer C; Institute of Musculoskeletal Medicine, University Hospital Münster, Albert-Schweitzer-Campus 1, Building D3, Münster, Germany.
Intemann J; Institute of Musculoskeletal Medicine, University Hospital Münster, Albert-Schweitzer-Campus 1, Building D3, Münster, Germany.
de Gorter DJJ; Institute of Musculoskeletal Medicine, University Hospital Münster, Albert-Schweitzer-Campus 1, Building D3, Münster, Germany.
Dankbar B; Institute of Musculoskeletal Medicine, University Hospital Münster, Albert-Schweitzer-Campus 1, Building D3, Münster, Germany.
Hillen J; Institute of Musculoskeletal Medicine, University Hospital Münster, Albert-Schweitzer-Campus 1, Building D3, Münster, Germany.
Heitzmann M; Institute of Musculoskeletal Medicine, University Hospital Münster, Albert-Schweitzer-Campus 1, Building D3, Münster, Germany.
Begemann I; Institute of Medical Physics and Biophysics, University of Münster, Robert-Koch Straße 31, Münster, Germany.
Galic M; Institute of Medical Physics and Biophysics, University of Münster, Robert-Koch Straße 31, Münster, Germany.
Weinhage T; Department of Pediatric Rheumatology and Immunology, University Children's Hospital Münster, Domagkstraße 3, Münster, Germany.
Foell D; Department of Pediatric Rheumatology and Immunology, University Children's Hospital Münster, Domagkstraße 3, Münster, Germany.
Ai R; Department of Chemistry and Biochemistry, 9500 Gilman Drive, UC San Diego, La Jolla, CA, USA.
Kremerskothen J; Department of Nephrology and Rheumatology, Internal Medicine D, University Hospital Münster, Albert-Schweitzer-Campus 1, Münster, Germany.
Kiener HP; Department of Medicine III, Division of Rheumatology, Medical University of Vienna, Waehringer Guertel 18-20, Vienna, Austria.
Müller S; Institute for Immunology, Jena University Hospital, Friedrich Schiller University, Jena, Leutragraben 3, Jena, Germany.
Kamradt T; Institute for Immunology, Jena University Hospital, Friedrich Schiller University, Jena, Leutragraben 3, Jena, Germany.
Schröder C; Genome Informatics, Institute of Human Genetics, University of Duisburg-Essen, Virchowstraße 183, Essen, Germany.
Leitão E; Institute of Human Genetics, University Hospital of Essen, University of Duisburg-Essen, Hufelandstraße 55, Essen, Germany.
Horsthemke B; Institute of Human Genetics, University Hospital of Essen, University of Duisburg-Essen, Hufelandstraße 55, Essen, Germany.
Rosenstiel P; Institute of Clinical Molecular Biology, University of Kiel, Rosalind-Franklin-Straße 12, Kiel, Germany.
Nordström K; Department of Genetics/Epigenetics, Saarland University, Campus Saarbrücken, Building A2 4, Saarbrücken, Germany.
Gasparoni G; Department of Genetics/Epigenetics, Saarland University, Campus Saarbrücken, Building A2 4, Saarbrücken, Germany.
Gasparoni N; Department of Genetics/Epigenetics, Saarland University, Campus Saarbrücken, Building A2 4, Saarbrücken, Germany.
Walter J; Department of Genetics/Epigenetics, Saarland University, Campus Saarbrücken, Building A2 4, Saarbrücken, Germany.
Li N; Max Planck Institute for Molecular Genetics, Otto-Warburg-Laboratories, Epigenomics, Ihnestraße 63-73, Berlin, Germany.
Yang X; Max Planck Institute for Molecular Genetics, Otto-Warburg-Laboratories, Epigenomics, Ihnestraße 63-73, Berlin, Germany.
Chung HR; Max Planck Institute for Molecular Genetics, Otto-Warburg-Laboratories, Epigenomics, Ihnestraße 63-73, Berlin, Germany.; Institute of Medical Bioinformatics and Biostatistics, Philipps-University Marburg, Bunsenstraße 3, Marburg, Germany.
Pavenstädt H; Department of Nephrology and Rheumatology, Internal Medicine D, University Hospital Münster, Albert-Schweitzer-Campus 1, Münster, Germany.
Lindemann N; Institute of Anatomy and Vascular Biology, University of Münster, Vesaliusweg 2-4, Münster, Germany.
Schnittler HJ; Institute of Anatomy and Vascular Biology, University of Münster, Vesaliusweg 2-4, Münster, Germany.
Wang W; Department of Chemistry and Biochemistry, 9500 Gilman Drive, UC San Diego, La Jolla, CA, USA.; Department of Cellular and Molecular Medicine, 9500 Gilman Drive, UCSD School of Medicine, La Jolla, CA, USA.
Firestein GS; Division of Rheumatology, Allergy and Immunology, 9500 Gilman Drive, UCSD School of Medicine, La Jolla, CA, USA.
Pap T; Institute of Musculoskeletal Medicine, University Hospital Münster, Albert-Schweitzer-Campus 1, Building D3, Münster, Germany.
Korb-Pap A; Institute of Musculoskeletal Medicine, University Hospital Münster, Albert-Schweitzer-Campus 1, Building D3, Münster, Germany. .
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Źródło :
Nature communications [Nat Commun] 2021 Jun 15; Vol. 12 (1), pp. 3624. Date of Electronic Publication: 2021 Jun 15.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Adaptor Proteins, Signal Transducing/*metabolism
Adherens Junctions/*metabolism
Arthritis/*metabolism
Cell Transformation, Neoplastic/*metabolism
Cytoskeletal Proteins/*metabolism
Fibroblasts/*metabolism
LIM Domain Proteins/*metabolism
Adaptor Proteins, Signal Transducing/genetics ; Animals ; Arthritis/pathology ; Arthritis, Rheumatoid/metabolism ; Arthritis, Rheumatoid/pathology ; Cadherins/metabolism ; Cytoskeletal Proteins/genetics ; Female ; Homeodomain Proteins ; LIM Domain Proteins/genetics ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Osteoblasts ; beta Catenin/metabolism
Czasopismo naukowe
Tytuł :
Glaucocalyxin A suppresses inflammatory responses and induces apoptosis in TNF-a-induced human rheumatoid arthritis via modulation of the STAT3 pathway.
Autorzy :
Zhang C; Orthopedic Hospital of Henan Province & Orthopedic Institute of Henan Province, Luoyang, Henan, 471002, PR China.
Ma K; Orthopedic Hospital of Henan Province & Orthopedic Institute of Henan Province, Luoyang, Henan, 471002, PR China.
Yang Y; Orthopedic Hospital of Henan Province & Orthopedic Institute of Henan Province, Luoyang, Henan, 471002, PR China.
Wang F; Anyang Cancer Hospital, Anyang, Henan, 455000, PR China.
Li W; Orthopedic Hospital of Henan Province & Orthopedic Institute of Henan Province, Luoyang, Henan, 471002, PR China. Electronic address: .
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Źródło :
Chemico-biological interactions [Chem Biol Interact] 2021 May 25; Vol. 341, pp. 109451. Date of Electronic Publication: 2021 Mar 30.
Typ publikacji :
Journal Article
MeSH Terms :
Arthritis, Rheumatoid/*drug therapy
Diterpenes, Kaurane/*pharmacology
STAT3 Transcription Factor/*metabolism
Animals ; Anti-Inflammatory Agents, Non-Steroidal/pharmacology ; Apoptosis/drug effects ; Arthritis, Experimental/drug therapy ; Arthritis, Experimental/pathology ; Arthritis, Rheumatoid/metabolism ; Arthritis, Rheumatoid/pathology ; CD4-Positive T-Lymphocytes/drug effects ; Cell Differentiation/drug effects ; Cell Proliferation/drug effects ; Cells, Cultured ; Cytokines/metabolism ; Humans ; Inflammation/drug therapy ; Inflammation/metabolism ; Inflammation/pathology ; Male ; Mice, Inbred DBA ; Rats, Wistar ; STAT3 Transcription Factor/genetics ; Synoviocytes/drug effects ; Synoviocytes/metabolism ; Synoviocytes/pathology ; Th17 Cells/drug effects ; Th17 Cells/physiology ; Tumor Necrosis Factor-alpha/pharmacology
Czasopismo naukowe
Tytuł :
Axial psoriatic arthritis: An update for dermatologists.
Autorzy :
Gottlieb AB; Icahn School of Medicine at Mt Sinai, New York, New York. Electronic address: .
Merola JF; Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts.
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Źródło :
Journal of the American Academy of Dermatology [J Am Acad Dermatol] 2021 Jan; Vol. 84 (1), pp. 92-101. Date of Electronic Publication: 2020 Jul 31.
Typ publikacji :
Journal Article; Review
MeSH Terms :
Arthritis, Psoriatic/*diagnosis
Arthritis, Psoriatic/*drug therapy
Antirheumatic Agents/therapeutic use ; Arthritis, Juvenile/complications ; Arthritis, Juvenile/diagnosis ; Arthritis, Juvenile/drug therapy ; Arthritis, Psoriatic/complications ; Arthritis, Psoriatic/immunology ; Early Diagnosis ; Humans ; Quality of Life ; Risk Factors
Czasopismo naukowe
Tytuł :
Disease Differentiation and Monitoring of Anti-TNF Treatment in Rheumatoid Arthritis and Spondyloarthropathies.
Autorzy :
Bogunia-Kubik K; Laboratory of Clinical Immunogenetics and Pharmacogenetics, Hirszfeld Institute of Immunology and Experimental Therapy, Polish Academy of Sciences, 53-114 Wroclaw, Poland.
Wojtowicz W; Department of Bioorganic Chemistry, Wroclaw University of Technology, 50-370 Wroclaw, Poland.
Swierkot J; Department of Rheumatology and Internal Medicine, Wroclaw Medical University, 50-556 Wroclaw, Poland.
Mielko KA; Department of Bioorganic Chemistry, Wroclaw University of Technology, 50-370 Wroclaw, Poland.
Qasem B; Department of Bioorganic Chemistry, Wroclaw University of Technology, 50-370 Wroclaw, Poland.
Wielińska J; Laboratory of Clinical Immunogenetics and Pharmacogenetics, Hirszfeld Institute of Immunology and Experimental Therapy, Polish Academy of Sciences, 53-114 Wroclaw, Poland.
Sokolik R; Department of Rheumatology and Internal Medicine, Wroclaw Medical University, 50-556 Wroclaw, Poland.
Pruss Ł; Department of Bioorganic Chemistry, Wroclaw University of Technology, 50-370 Wroclaw, Poland.; Ardigen, 30-394 Krakow, Poland.
Młynarz P; Department of Bioorganic Chemistry, Wroclaw University of Technology, 50-370 Wroclaw, Poland.
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Źródło :
International journal of molecular sciences [Int J Mol Sci] 2021 Jul 09; Vol. 22 (14). Date of Electronic Publication: 2021 Jul 09.
Typ publikacji :
Journal Article
MeSH Terms :
Arthritis, Psoriatic/*blood
Arthritis, Rheumatoid/*blood
Ethanol/*blood
Spondylitis, Ankylosing/*blood
Tumor Necrosis Factor Inhibitors/*therapeutic use
Adult ; Arthritis, Psoriatic/drug therapy ; Arthritis, Rheumatoid/drug therapy ; Cohort Studies ; Computational Biology ; Female ; Humans ; Magnetic Resonance Spectroscopy ; Male ; Metabolome ; Principal Component Analysis ; Spondylitis, Ankylosing/drug therapy
Czasopismo naukowe
Tytuł :
Lipoxin A4-Mediated p38 MAPK Signaling Pathway Protects Mice Against Collagen-Induced Arthritis.
Autorzy :
Li J; Department of Orthopedic, Dongzhimen Hospital, Beijing University of Chinese Medicine, No. 5 Hai Yun Cang, Dong Cheng District, Beijing, 100700, China.
Sun Q; Department of Orthopedic, Dongzhimen Hospital, Beijing University of Chinese Medicine, No. 5 Hai Yun Cang, Dong Cheng District, Beijing, 100700, China.
Zheng C; Department of Orthopedic, Dongzhimen Hospital, Beijing University of Chinese Medicine, No. 5 Hai Yun Cang, Dong Cheng District, Beijing, 100700, China.
Bai C; Department of Orthopedic, Dongzhimen Hospital, Beijing University of Chinese Medicine, No. 5 Hai Yun Cang, Dong Cheng District, Beijing, 100700, China.
Liu C; Department of Orthopedic, Dongzhimen Hospital, Beijing University of Chinese Medicine, No. 5 Hai Yun Cang, Dong Cheng District, Beijing, 100700, China.
Zhao X; Department of Orthopedic, Dongzhimen Hospital, Beijing University of Chinese Medicine, No. 5 Hai Yun Cang, Dong Cheng District, Beijing, 100700, China.
Deng P; Key Laboratory of Chinese Internal Medicine of Ministry of Education and Beijing, Dongzhimen Hospital, Beijing University of Chinese Medicine, Beijing, 100700, China.
Chai L; Key Laboratory of Chinese Internal Medicine of Ministry of Education and Beijing, Dongzhimen Hospital, Beijing University of Chinese Medicine, Beijing, 100700, China. .
Jia Y; Department of Orthopedic, Dongzhimen Hospital, Beijing University of Chinese Medicine, No. 5 Hai Yun Cang, Dong Cheng District, Beijing, 100700, China. .
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Źródło :
Biochemical genetics [Biochem Genet] 2021 Feb; Vol. 59 (1), pp. 346-365. Date of Electronic Publication: 2020 Nov 22.
Typ publikacji :
Journal Article
MeSH Terms :
MAP Kinase Signaling System*
Arthritis, Rheumatoid/*metabolism
Collagen/*metabolism
Lipoxins/*pharmacology
p38 Mitogen-Activated Protein Kinases/*metabolism
Animals ; Anisomycin/pharmacology ; Arthritis, Experimental/chemically induced ; Arthritis, Experimental/metabolism ; Arthritis, Experimental/prevention & control ; Arthritis, Rheumatoid/prevention & control ; Cell Proliferation ; Female ; Imidazoles/pharmacology ; Inflammation ; Mice ; Mice, Inbred DBA ; Oxidative Stress ; Pyridines/pharmacology ; Signal Transduction ; Synovial Membrane/metabolism
Czasopismo naukowe
Tytuł :
Acid-Sensing Ion Channel-1a in Articular Chondrocytes and Synovial Fibroblasts: A Novel Therapeutic Target for Rheumatoid Arthritis.
Autorzy :
Xu Y; Department of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, China.; The Key Laboratory of Major Autoimmune Diseases of Anhui Province, Anhui Institute of Innovative Drugs, School of Pharmacy, Anhui Medical University, Hefei, China.; The Key Laboratory of Anti-inflammatory and Immune Medicines, Ministry of Education, Hefei, China.
Chen F; Department of Epidemiology and Biostatistics, School of Public Health, Anhui Medical University, Hefei, China.; The Key Laboratory of Major Autoimmune Diseases of Anhui Province, Anhui Institute of Innovative Drugs, School of Pharmacy, Anhui Medical University, Hefei, China.; The Key Laboratory of Anti-inflammatory and Immune Medicines, Ministry of Education, Hefei, China.
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Źródło :
Frontiers in immunology [Front Immunol] 2021 Jan 28; Vol. 11, pp. 580936. Date of Electronic Publication: 2021 Jan 28 (Print Publication: 2020).
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't; Review
MeSH Terms :
Acid Sensing Ion Channel Blockers/*pharmacology
Acid Sensing Ion Channels/*metabolism
Arthritis, Rheumatoid/*drug therapy
Arthritis, Rheumatoid/*metabolism
Cartilage, Articular/*metabolism
Chondrocytes/*metabolism
Synovial Membrane/*metabolism
Animals ; Arthritis, Experimental/drug therapy ; Arthritis, Experimental/metabolism ; Arthritis, Experimental/pathology ; Arthritis, Rheumatoid/pathology ; Cartilage, Articular/pathology ; Chondrocytes/pathology ; Fibroblasts/metabolism ; Fibroblasts/pathology ; Humans ; Models, Biological ; Rats ; Regulated Cell Death/physiology ; Signal Transduction ; Synovial Membrane/pathology
Czasopismo naukowe
Tytuł :
Th17 lymphocyte-dependent degradation of joint cartilage by synovial fibroblasts in a humanized mouse model of arthritis and reversal by secukinumab.
Autorzy :
Margheri F; Department of Experimental and Clinical Biomedical Sciences 'Mario Serio', University of Florence, Florence, Italy.
Maggi L; Department of Experimental and Clinical Medicine, University of Florence, Florence, Italy.
Biagioni A; Department of Experimental and Clinical Biomedical Sciences 'Mario Serio', University of Florence, Florence, Italy.
Chillà A; Department of Experimental and Clinical Biomedical Sciences 'Mario Serio', University of Florence, Florence, Italy.
Laurenzana A; Department of Experimental and Clinical Biomedical Sciences 'Mario Serio', University of Florence, Florence, Italy.
Bianchini F; Department of Experimental and Clinical Biomedical Sciences 'Mario Serio', University of Florence, Florence, Italy.
Bani D; Department of Experimental and Clinical Medicine, University of Florence, Florence, Italy.
Capone M; Department of Experimental and Clinical Medicine, University of Florence, Florence, Italy.
Mazzoni A; Department of Experimental and Clinical Medicine, University of Florence, Florence, Italy.
Rossi MC; Department of Experimental and Clinical Medicine, University of Florence, Florence, Italy.
Liotta F; Department of Experimental and Clinical Medicine, University of Florence, Florence, Italy.
Cosmi L; Department of Experimental and Clinical Medicine, University of Florence, Florence, Italy.
Giani T; AOU Meyer, Florence, Italy.
Cimaz R; Department of Clinical Sciences and Community Health, University of Milano, Milan, Italy.
Fibbi G; Department of Experimental and Clinical Biomedical Sciences 'Mario Serio', University of Florence, Florence, Italy.
Annunziato F; Department of Experimental and Clinical Medicine, University of Florence, Florence, Italy.
Del Rosso M; Department of Experimental and Clinical Biomedical Sciences 'Mario Serio', University of Florence, Florence, Italy.
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Źródło :
European journal of immunology [Eur J Immunol] 2021 Jan; Vol. 51 (1), pp. 220-230. Date of Electronic Publication: 2020 Aug 09.
Typ publikacji :
Journal Article; Research Support, Non-U.S. Gov't
MeSH Terms :
Antibodies, Monoclonal, Humanized/*therapeutic use
Arthritis, Juvenile/*immunology
Arthritis, Juvenile/*therapy
Cartilage, Articular/*immunology
Cartilage, Articular/*pathology
Th17 Cells/*immunology
Th17 Cells/*pathology
Adolescent ; Animals ; Arthritis, Experimental/immunology ; Arthritis, Experimental/pathology ; Arthritis, Experimental/therapy ; Arthritis, Juvenile/pathology ; Cartilage, Articular/metabolism ; Case-Control Studies ; Child ; Child, Preschool ; Cytokines/immunology ; Disease Models, Animal ; Fibroblasts/immunology ; Fibroblasts/metabolism ; Fibroblasts/pathology ; Humans ; In Vitro Techniques ; Interleukin-17/antagonists & inhibitors ; Mice ; Mice, SCID ; Proteolysis ; Synovial Membrane/immunology ; Synovial Membrane/metabolism ; Synovial Membrane/pathology
Czasopismo naukowe

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